Concussion and CTE

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If CTE progresses long enough, the patient’s brain exhibits actual physical atrophy, much like the brains of frontotemporal dementia or Alzheimer’s patients.


CTE occurs when repetitive head trauma begins to produce abnormal proteins in the brain known as “tau.”


Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis PP2A to decrease phosphorylation at T34 and increase phosphorylation at T75.


What Does Tau Protein Have to Do with Football, Dementia, and Suicide? (PP2A regulates TAU Protien).

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